{"id":1206,"date":"2025-10-24T11:09:07","date_gmt":"2025-10-24T15:09:07","guid":{"rendered":"https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA.jpg"},"modified":"2026-07-29T12:03:26","modified_gmt":"2026-07-29T16:03:26","slug":"sharon-yaqoob-krysztofiak","status":"inherit","type":"attachment","link":"https:\/\/health.uconn.edu\/molecular-oncology\/sharon-yaqoob-krysztofiak\/","title":{"rendered":"Sharon Yaqoob-Krysztofiak (pictured here with Dr. Andrew Arnold) co-authored \u201cInvestigating the in Vivo Role of an Activating GCM2 Variant, I383M, as a Potential Contributor to the Pathogenesis of Primary Hyperparathyroidism\u201d at the July 2025 Endocrine Society Annual Meeting in San Francisco, CA."},"author":2565,"featured_media":0,"comment_status":"open","ping_status":"closed","template":"","meta":{"_acf_changed":false},"acf":[],"smush":{"stats":{"time":0.42000000000000004,"bytes":90281,"percent":5.28,"size_before":1710296,"size_after":1620015,"lossy":0,"keep_exif":0,"api_version":"1.0"},"sizes":{"sow-carousel-default":{"time":0.01,"bytes":615,"percent":4.57,"size_before":13469,"size_after":12854},"medium":{"time":0.01,"bytes":788,"percent":4.48,"size_before":17600,"size_after":16812},"thumbnail":{"time":0.03,"bytes":391,"percent":5.23,"size_before":7479,"size_after":7088},"medium_large":{"time":0.03,"bytes":3295,"percent":3.84,"size_before":85768,"size_after":82473},"large":{"time":0.03,"bytes":5502,"percent":3.92,"size_before":140397,"size_after":134895},"1536x1536":{"time":0.05,"bytes":13087,"percent":4.65,"size_before":281362,"size_after":268275},"2048x2048":{"time":0.1,"bytes":24854,"percent":5.33,"size_before":466048,"size_after":441194},"wp_scaled":{"time":0.16,"bytes":41749,"percent":5.98,"size_before":698173,"size_after":656424}}},"description":{"rendered":"<p class=\"attachment\"><a href='https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-scaled.jpg'><img loading=\"lazy\" decoding=\"async\" width=\"300\" height=\"225\" src=\"https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-300x225.jpg\" class=\"attachment-medium size-medium\" alt=\"Sharon Yaqoob-Krysztofiak (pictured here with Dr. Andrew Arnold) co-authored \u201cInvestigating the in Vivo Role of an Activating GCM2 Variant, I383M, as a Potential Contributor to the Pathogenesis of Primary Hyperparathyroidism\u201d at the July 2025 Endocrine Society Annual Meeting in San Francisco, CA.\" srcset=\"https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-300x225.jpg 300w, https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-1024x768.jpg 1024w, https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-768x576.jpg 768w, https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-1536x1152.jpg 1536w, https:\/\/health.uconn.edu\/molecular-oncology\/wp-content\/uploads\/sites\/116\/2025\/10\/Endo-Sharon-AA-2048x1536.jpg 2048w\" sizes=\"(max-width: 300px) 100vw, 300px\" \/><\/a><\/p>\n<p>Sharon Yaqoob-Krysztofiak (pictured here with Dr. Andrew Arnold) co-authored \u201cInvestigating the in Vivo Role of an Activating GCM2 Variant, I383M, as a Potential Contributor to the Pathogenesis of Primary Hyperparathyroidism\u201d at the July 2025 Endocrine Society Annual Meeting in San Francisco, CA.<\/p>\n"},"caption":{"rendered":"<p>Sharon Yaqoob-Krysztofiak (pictured here with Dr. Andrew Arnold) co-authored \u201cInvestigating the in Vivo Role of an Activating GCM2 Variant, I383M, as a Potential Contributor to the Pathogenesis of Primary Hyperparathyroidism\u201d at the July 2025 Endocrine Society Annual Meeting in San Francisco, CA.<\/p>\n"},"alt_text":"Sharon Yaqoob-Krysztofiak (pictured here with Dr. Andrew Arnold) co-authored \u201cInvestigating the in Vivo Role of an Activating GCM2 Variant, I383M, as a Potential Contributor to the Pathogenesis of Primary Hyperparathyroidism\u201d at the July 2025 Endocrine Society Annual Meeting in San Francisco, 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